Longevity & Aging Series (S1, E2): Dr. Steve Horvath's Special Collection in Aging | Aging-US
In the second installment of the Longevity & Aging Series, Dr. Steve Horvath, Professor of Human Genetics and Biostatistics at UCLA, and Principal Investigator at Altos Labs, revisits papers he co-authored that were published by Aging (Aging-US) and featured as a special collection of his research.
#aging #interview
Special Collection - https://www.aging-us.com/special-coll...
Author contact - Steve Horvath - [email protected]
Papers -
DNA-methylation-based telomere length estimator: comparisons with measurements from flow FISH and qPCR
https://doi.org/10.18632/aging.203126
Epigenetic mutation load is weakly correlated with epigenetic age acceleration
https://doi.org/10.18632/aging.103950
Blood DNA methylation sites predict death risk in a longitudinal study of 12, 300 individuals
https://doi.org/10.18632/aging.103408
Epigenome-wide association study of leukocyte telomere length
https://doi.org/10.18632/aging.102230
DNA methylation-based estimator of telomere length
https://doi.org/10.18632/aging.102173
Optimism is not associated with two indicators of DNA methylation aging
https://doi.org/10.18632/aging.102090
Placental epigenetic clocks: estimating gestational age using placental DNA methylation levels
https://doi.org/10.18632/aging.102049
Rapamycin retards epigenetic ageing of keratinocytes independently of its effects on replicative senescence, proliferation and differentiation
https://doi.org/10.18632/aging.101976
Epigenetic clock analysis of human fibroblasts in vitro: effects of hypoxia, donor age, and expression of hTERT and SV40 largeT
https://doi.org/10.18632/aging.101955
DNA methylation GrimAge strongly predicts lifespan and healthspan
https://doi.org/10.18632/aging.101684
Cell and tissue type independent age-associated DNA methylation changes are not rare but common
https://doi.org/10.18632/aging.101666
A multi-tissue full lifespan epigenetic clock for mice
https://doi.org/10.18632/aging.101590
Epigenetic ageing is distinct from senescence-mediated ageing and is not prevented by telomerase expression
https://doi.org/10.18632/aging.101588
Epigenetic clock for skin and blood cells applied to Hutchinson Gilford Progeria Syndrome and ex vivo studies
https://doi.org/10.18632/aging.101508
An epigenetic biomarker of aging for lifespan and healthspan
https://doi.org/10.18632/aging.101414
Leukocyte telomere length, T cell composition and DNA methylation age
https://doi.org/10.18632/aging.101293
Accelerated epigenetic aging in Werner syndrome
https://doi.org/10.18632/aging.101217
An epigenetic aging clock for dogs and wolves
https://doi.org/10.18632/aging.101211
Longitudinal study of surrogate aging measures during human immunodeficiency virus seroconversion
https://doi.org/10.18632/aging.101184
Epigenetic clock analysis of diet, exercise, education, and lifestyle factors
https://doi.org/10.18632/aging.101168
Specific premature epigenetic aging of cartilage in osteoarthritis
https://doi.org/10.18632/aging.101053
DNA methylation-based measures of biological age: meta-analysis predicting time to death
https://doi.org/10.18632/aging.101020
Huntington's disease accelerates epigenetic aging of human brain and disrupts DNA methylation levels
https://doi.org/10.18632/aging.101005
Epigenetic age of the pre-frontal cortex is associated with neuritic plaques, amyloid load, and Alzheimer’s disease related cognitive functioning
https://doi.org/10.18632/aging.100864
Decreased epigenetic age of PBMCs from Italian semi-supercentenarians and their offspring
https://doi.org/10.18632/aging.100861
Increased epigenetic age and granulocyte counts in the blood of Parkinson's disease patients
https://doi.org/10.18632/aging.100859
DNA methylation age of blood predicts future onset of lung cancer in the women's health initiative
https://doi.org/10.18632/aging.100809
Epigenetic age analysis of children who seem to evade aging
https://doi.org/10.18632/aging.100744
The cerebellum ages slowly according to the epigenetic clock
https://doi.org/10.18632/aging.100742
About Aging-US
Launched in 2009, Aging-US publishes papers of general interest and biological significance in all fields of aging research and age-related diseases, including cancer—and now, with a special focus on COVID-19 vulnerability as an age-dependent syndrome. Topics in Aging-US go beyond traditional gerontology, including, but not limited to, cellular and molecular biology, human age-related diseases, pathology in model organisms, signal transduction pathways (e.g., p53, sirtuins, and PI-3K/AKT/mTOR, among others), and approaches to modulating these signaling pathways.
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